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| This study is a case-control GWAS comprised of 1393 SLE cases and 886 controls. All samples were genotyped on the HumanOmni1-Quad BeadChip using manifest H at the Oklahoma Medical Research Foundation. The Illumina clustering algorithm with GenomeStudio v2011.1 was used. SLE was the trait of interest (2=SLE Case, 1=Control), and we adjusted for two principal components. |